Exercise-induced ­bronchoconstriction­ is­ defined­ as­ reversible­ narrowing­ of­ distal­ airways that­ follows­ vigorous exercise­ in­ the­ presence­ or­ absence­ of­ clinically­ recognised­ asthma.­ The­ prevalence­ in­ general­ population­ is­ 7–20%, in­ athletes­ even­ higher,­ around­ 50%. Water ­depletion­ at­ the­ level­ of­ airways­ is­ a­ reaction­ to­ increased­ minute ventilation­ and represents­ a­ stimulus­ to­ exercise-induced­ bronchoconstriction.­ There­ are­ two­ theories seeking­ to­ explain­ exercise-induced­ bronchoconstriction:­ thermal­ and­ osmotic.­ In­ the­ laboratory­ conditions,­ the­ establishment of­ the­ diagnosis­ can­ be­ difficult.­ The­ type­ of­ protocol­ used­ for­ diagnosis­ must­ consider­ mechanistic­ factors­ for exercise-induced­ bronchoconstriction.­ To­ elicit­ dehydration­ of­ the­ airway­ surface­ liquid­ and­ to­ cause­ a­ transient increase­ in­ its­ osmolarity,­ the­ rate­ of­ water­ loss­ must­ exceed­ the­ rate­ of­ return­ in­ the first­ 10­ generations­ of­ airways. In­ general,­ the­ drier­ the­ inspired­ air­ and­ the­ higher­ the ventilation­ sustained­ during­ exercise,­ the­ less­ likely­ a­ false negative­ test­ result­ will­ occur. There­ are­ two­ forms­ of­ bronchoprovocation:­ direct­ and­ indirect.­ They­ differ­ with respect to­ basic­ mechanisms­ of­ inducing­ bronchoprovocation.­ The­ management­ of­ exercise-induced bronchoconstriction­ should­ include­ both­ prevention­ and­ treatment­ directed­ toward­ the underlying­ asthma­ and­ bronchial­ hyperresponsiveness.­ Among­ the­ nonfarmacological components­ of­ treatment,­ high­ intensity­ warm­ up­ is recommended.